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Wang Guihua[1,2] *

; Long Jie[2,3] ; Gao Yuan[4] ; Zhang Weina[2] ; Han Fei[2] ; Xu Chuan[2] ; Sun Li[1,2] ; Yang Shun-Chin[2] ; Lan Jingqin[1]

; Hou Zhenlin[1] ; Cai Zhen[2] ; Jin Guoxiang[2] ; Hsu Che-Chia[2] ; Wang Yu-Hui[2] ; Hu Junbo[1]

; Chen Tsai-Yu[4] ; Li Hongyu[5] ; Lee Min Gyu[4] ; Lin Hui-Kuan[6,7,8] ;

单位： [1]Department of GI Cancer Research Institute, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, Hubei, China. [2]Department of Cancer Biology, Wake Forest Baptist Medical Center, Wake Forest University, Winston Salem, NC, USA. [3]Department of Pathology, School of Basic Medical Science, Guangzhou Medical University, Guangzhou, Guangdong, China. [4]Department of Molecular and Cellular Oncology, The University of Texas MD Anderson Cancer Center, Houston, TX, USA. [5]University of Arkansas for Medical Sciences, College of Pharmacy, Department of Pharmaceutical Science, Little Rock, AR, USA. [6]Department of Cancer Biology, Wake Forest Baptist Medical Center, Wake Forest University, Winston Salem, NC, USA. hulin@wakehealth.edu. [7]Graduate Institute of Basic Medical Science, China Medical University, Taichung, Taiwan. hulin@wakehealth.edu. [8]Department of Biotechnology, Asia University, Taichung, Taiwan. hulin@wakehealth.edu.

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出版当年[2020]版：

大类 | 1 区

生物

小类 | 1 区细胞生物学

最新[2025]版：

大类 | 1 区

生物学

小类 | 1 区细胞生物学

第一作者：

第一作者单位： [1]Department of GI Cancer Research Institute, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, Hubei, China. [2]Department of Cancer Biology, Wake Forest Baptist Medical Center, Wake Forest University, Winston Salem, NC, USA.

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Author Correction: SETDB1-mediated methylation of Akt promotes its K63-linked ubiquitination and activation leading to tumorigenesis.

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